The use of neuromuscular blocking drugs carries with it a very small risk of anesthesia awareness.
2.
If neuromuscular blocking drugs are used this causes skeletal muscle paralysis and interferes with the functioning of the autonomic nervous system.
3.
This is very unusual with all but the longest acting neuromuscular blocking drugs ( such as gallamine, pancuronium or tubocurarine ).
4.
They are thought to be the chemical group responsible for anaphylactic reactions that occur with use of neuromuscular blocking drugs during general anaesthesia in surgery.
5.
'Recurarisation', a phenomenon of recurrence of neuromuscular block, may occur where the reversal agents wear off before a neuromuscular blocking drug is completely cleared.
6.
Mivacurium, a non-depolarizing neuromuscular blocking drug, is also metabolized via the same route with a similar clinical effect in patients deficient in plasma cholinesterase activity.
7.
The holy grail of research in the neuromuscular blocking drugs arena for the better part of the 1980s and 1990s has been to find a non-depolarizing replacement for succinylcholine.
8.
Then, a triple dose of a non-depolarizing neuromuscular blocking drug is given, such as 20 mg pancuronium bromide ( Pavulon ) or 20 mg vecuronium bromide ( Norcuron ).
9.
In this sense, gantacurium is a first in its class non-depolarizing neuromuscular blocking drug to arguably challenge the pharmacological profile of the gold-standard ultrashort acting depolarizing agent succinylcholine ( suxamethonium ).
10.
Typically, anesthesia is'induced'with an intravenous drug, but'maintained'with an neuromuscular blocking drug may then be given to cause paralysis which facilitates intubation, although this is not always required.
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